certified neurodiversity practitioner
neuroscience based regulation support
lifestyle medicine
The journey to health and restoration is a returning.
RINKA SYCIP, M.TH., IPHM
Integrative Health & Lifestyle Medicine Practitioner · Neuroscience-Based Coaching · Certified Neurodiversity Practitioner
Rinka SyCip is an Integrative Health and Lifestyle Medicine Practitioner. Her practice is neuroscience-based performance and wellness coaching.She is a Certified Neuroscience and Neurodiversity Practitioner (ADHD / Autism / AuDHD), certified by the International Practitioners of Holistic Medicine (IPHM, UK) and the International Association of Therapists (IAOTH).She holds a Master’s in Theology and a certificate in Lifestyle Medicine Coaching from Harvard Medical School.She was also awarded a Merit Scholarship to an Advanced Lifestyle Oncology program under the scientific sponsorship of MASCC (Multinational Association of Supportive Care in Cancer) and the International Cancer Rehabilitation Foundation (ICRF).These credentials formalize a path she has walked for years.In 2015, before any formal training, she and her husband — both late-diagnosed Autistic and ADHD — built their own stability toolkit while navigating chronic stress, burnout cycles, energy collapse, and nervous system volatility.In helping themselves and immediate family members manage chronic illness as well, the practice grew organically out of what helped those closest to her — refined in real households, under real conditions, long before it had a name.She works with late-diagnosed adults, families of neurodivergent individuals, and professionals who function at a high level yet live near burnout.She supportsLate-diagnosed or self-identified neurodivergent adults in burnout, shutdown, or cognitive drain, parents and families seeking non-shaming ways to understand and support neurodivergent loved onesExecutives, creatives, and knowledge workers who have relied on masking as their primary adaptationIndividuals carrying chronic stress, chronic illness, or systemic symptoms unresolved by standard care.Clients often arrive after exhausting traditional options.Coaching is customized, longitudinal, and structured to hold through flare-ups, meltdowns, and chronic stress — without forcing neurotypical assumptions onto neurodivergent lives.Every engagement moves through Regulate → Scaffold → Sustain: first the nervous system finds its footing, then structures are built around what works, then the gains become a way of living.Her background in theology informs the way she works with meaning, purpose, and internal orientation. It adds a layer to coaching that accounts for spiritual coherence, personal conviction, integrity, and the deeper narratives that shape resilience.
Scoping Session
The scoping session is a crucial one. Here we put together a needs analysis and craft a coaching plan for the client. This way, we can move forward with well-defined, achievable and actionable goals together. This runs for 60-90 minutes.For non-neurodivergent individuals, coaching sessions focus on neuroscience-based approaches to nervous system regulation, and stress resilience.This includes assessment and reports on physical symptom mapping as well as preliminary neurodiversity screening for high-masking Autism, ADD/ADHD as well as AuDHD. (Note: Note a diagnosis, but a good first step toward official diagnosis)Online on Zoom
Standard Follow-Up Session
After a first intake session, book a follow-up session. Regular (biweekly, weekly or monthly) helps ensure progress and prevents nervous system overload.Can be individual, couple, or parent and child session as well.
60 minute session, online on Zoom
Group Session
For groups of four or more, we can do a nervous system regulation brown bag session. Minimum of 120 minutes, on site or on zoom.
Book for families, communities and teams. Faith-neutral or faith-anchored, bespoke programs for you. Leave a message to inquire.
For advocacy groups and NGOs, pro bono is available.
Corporate Workshop: From Fight Or Flight to Flow
For corporate organizations, high-stress, high-stakes, high-pressure teams, we offer workshops through our partner, OTI Group.
PEPTIDE THERAPY
Your body may be missing some essential amino acids, your body's building blocks, which is why some processes are not working optimally. Digestion, recovery, metabolism -- all these things and more can be profoundly impacted by these missing "building blocks". Click on the buttons below to learn more about them.
5AMINO1MQ is a powerful weight loss peptide, working more on the optimization of energy use.It plugs up unseen, unknown leaks that cause your body to lose NNMT (the amino acid that NAD+ re-supplies), and allows your body to retain and better utilize energy.
NAD+ is a potent anti-aging peptide. This is a naturally occurring coenzyme found in all living cells. It plays an essential role in converting nutrients into energy and supporting various cellular processes. It gets depleted as we age and are subjected to stress.
NAD+ is a potent anti-aging peptide. This is a naturally occurring coenzyme found in all living cells. It plays an essential role in converting nutrients into energy and supporting various cellular processes. It gets depleted as we age and are subjected to stress.
NAD+ (Nicotinamide Adenine Dinucleotide)
Nicotinamide adenine dinucleotide, commonly known as NAD+, is a naturally occurring coenzyme found in all living cells. It plays an essential role in converting nutrients into energy and supporting various cellular processes. NAD+ exists in two forms: oxidized NAD+ and reduced NADH. These forms work together to help transfer electrons during energy production, particularly in cellular respiration.NAD+ is involved in the breakdown of carbohydrates, fats, and proteins, allowing cells to produce adenosine triphosphate (ATP), the primary energy source of the body. It also supports enzymes called sirtuins, which are involved in cellular maintenance, stress responses, and the regulation of metabolism. Additionally, NAD+ is used by enzymes involved in DNA repair and maintaining cellular function.NAD+ levels naturally change with age, metabolic activity, and other biological factors. Because of its role in energy metabolism and cellular health, NAD+ has become a subject of interest in scientific research related to aging, exercise, and metabolic health. Researchers are studying whether increasing NAD+ availability through supplements or precursor compounds may influence these processes.NAD+ precursors, such as nicotinamide riboside (NR) and nicotinamide mononucleotide (NMN), are being investigated for their ability to support NAD+ production in the body. However, research is still ongoing, and the long-term benefits and safety of NAD+ supplementation in humans are not fully established.Overall, NAD+ is an important molecule that supports energy production, cellular repair, and normal biological function. Its broad role in metabolism makes it a significant focus of research in biochemistry, nutrition, and healthy aging.
5 AMINO 1MQ
5-amino-1MQ (5-amino-1methylquinolinium), similar to methylquinolinium, acts as a short peptide that blocks the cytosolic enzyme nicotinamide N-methyltransferase (NNMT). This enzyme connects to obesity and type 2 diabetes while helping control energy balance in cells. Blocking NNMT causes major weight loss, less fat mass and smaller fat cells (adipocytes), plus lower cholesterol and glucose in the blood. Researchers actively study 5-amino-1MQ and related methylquinolinium compounds as possible treatments for obesity and diabetes. Blocking NNMT also seems to activate stem cells and boost muscle repair capacity.ObesityNicotinamide N-methyltransferase serves as a cell enzyme present in many body areas, but it thrives most in liver and fat cells. Mouse research links high NNMT levels to lower amounts of the sugar transporter GLUT4. This transporter, found mainly in skeletal and heart muscle plus fat cells, ties closely to blood sugar control and diabetes risk. Rodent tests show mice with high GLUT4 production stay sensitive to insulin and resist type 2 diabetes better. In contrast, those with low GLUT4 face strong insulin resistance.GLUT4 connects to basic metabolic rate and ideas of fast versus slow metabolism. People with naturally high GLUT4 levels tend to have quicker metabolism and burn more calories. Exercise boosts this transporter's production, which helps explain how physical activity fights weight gain, high blood sugar, and insulin issues. GLUT4 and NNMT link tightly, affecting baseline metabolism in mammals. Dr. Barbara Kahn from Harvard Medical School notes that the GLUT4-NNMT tie first sparked interest in targeting this enzyme for diabetes and obesity care. Animals with insulin resistance often show high NNMT in fat cells. Adjusting this gene counters insulin resistance and thus diabetes, while strongly impacting weight and obesity.Human and animal metabolism runs with great efficiency, squeezing the most from few calories. Yet this same trait might drive obesity when calories abound. Making metabolism less efficient—causing the body to waste some calories—has long been a medical goal to tackle rising obesity. NNMT's role with GLUT4 could be the missing piece researchers seek. At its core, NNMT slows calorie use, leaving more for fat or glycogen storage. Lower NNMT cuts the shift of nicotinic acid (NA) to 1-methylnicotinamide (1-MNA). This change affects metabolism in two ways.Overall, using an NNMT blocker like 5-amino-1MQ ramps up energy burn and cuts storage. Paired with raised GLUT4 expression from lower NNMT, this setup clears glucose from blood better and uses it directly during burn. Outcomes include less insulin need, lower resistance, reduced fat buildup, and higher energy metabolism. In just 10 days, treated mice dropped 7% in weight and 30% in fat mass. Their blood cholesterol matched lean mice, all without food intake shifts. Mice on 5-amino-1MQ lost 7% body mass in 10 days without eating changes, unlike controls.Fresh evidence hints that 5-amino-1MQ's perks go beyond curbing NNMT and boosting inefficient metabolism plus GLUT4. Mouse studies suggest it reshapes fat cell function by raising GLUT4, prompting them to make different lipids with anti-diabetic and anti-inflammatory traits. These lipids, called PAHSAs (palmitic acid esters of hydroxy-stearic acids), ease insulin resistance alone and curb inflammation, improving odds against heart attacks and strokes. Though new, this side of 5-amino-1MQ suggests a wider range of benefits for this standout molecule.Muscle Function5-amino-1MQ affects skeletal muscle in several ways. Like in fat tissue, it boosts GLUT4 production there and raises metabolic inefficiency for more energy use. But recent mouse work shows inhibiting NNMT, including via 5-amino-1MQ, might speed muscle repair by sparking stem cells.Studies on 24-month-old mice (elderly for their kind) found those treated with an NNMT blocker had far more stem cell activation in hurt muscle than untreated ones. Their repaired muscle fibers grew twice as thick in cross-section and gained stronger contraction power. Treated mice showed 70% more force in healed muscle than controls. Beyond quickening injury fixes, boosting muscle stem cells could help older folks stay independent longer. By aiding movement and cutting fall risks, NNMT blockers might greatly enhance life quality and self-reliance in aging adults.Rising NNMT expression often marks muscle-wasting issues like Duchenne Muscular Dystrophy. Lowering NNMT could ease symptoms in some cases. This ties back to NNMT blocking stem cell growth and split. Compounds like 5-amino-1MQ, by dropping NNMT, might aid various muscle-loss conditions. NNMT block's full muscle effects aren't clear yet, but another angle involves NAD+ levels. Blocking NNMT raises NAD+, and tools like 5-amino-1MQ have boosted muscle work, heart health, and DMD in animal tests. Key drivers seem to be better mitochondrial function plus less inflammation and scarring, all from higher NAD+.CognitionNAD+ plays a vital part in brain energy balance. Low NAD+ links to many brain issues and harms neuron communication at synapses and nerve-muscle junctions. Mouse research shows falling NAD+ can weaken synaptic signals, hurt muscle, and harm overall thinking. While not tested directly here, 5-amino-1MQ likely affects brain NAD+ like elsewhere. This suggests it could treat thinking problems or act as a nootropic to sharpen average minds. Active interest explores 5-amino-1MQ's potential brain perks, though research must confirm.CancerStrong evidence shows higher NNMT in all gastric cancers, plus pancreatic, kidney, and bladder types. Mice lacking NNMT gene resist these cancers, hinting NNMT causes them. Early work speculates blocking NNMT could treat or prevent some cancers, or at least curb their spread. It's unclear if 5-amino-1MQ, by lowering NNMT, affects these cancers.
TIRZEPATIDE